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Parental Mental Health, Feeding Practices, and Sociodemographic Factors as Determinants of Childhood Obesity in Greece PDF Free Download

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Received: 23 December 2025
Revised: 11 January 2026
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Article
Parental Mental Health, Feeding Practices, and
Sociodemographic Factors as Determinants of Childhood
Obesity in Greece
Vlasia Stymfaliadi 1, Yannis Manios 2, Odysseas Androutsos 3, Maria Michou 1, Eleni Angelopoulou 4,
Xanthi Tigani 1, Panagiotis Pipelias 1, Styliani Katsouli 1and Christina Kanaka-Gantenbein 1,4,*
1Postgraduate Course “Science of Stress and Health Promotion”, Medical School, National and Kapodistrian
University of Athens (NKUA), 11527 Athens, Greece; vstymf@gmail.com (V.S.);
mariamixou@hotmail.com (M.M.); xanthitig@med.uoa.gr (X.T.); panospt@med.uoa.gr (P.P.);
stellakatsouli13@gmail.com (S.K.)
2Department of Nutrition and Dietetics, Harokopio University of Athens, 17676 Athens, Greece;
manios@hua.gr
3Laboratory of Nutrition & Clinical Dietetics, Department of Dietetics and Nutrition, School of Physical
Education, Sport and Dietetics, University of Thessaly, 42132 Trikala, Greece; oandroutsos@uth.gr
4First Department of Pediatrics, Medical School, National and Kapodistrian University of Athens,
“Aghia Sofia” Children’s Hospital, 11527 Athens, Greece; elen.angel@yahoo.gr
*Correspondence: ckanaka@med.uoa.gr or chriskan@med.uoa.gr
Abstract
Background/Objectives: Childhood obesity remains a major public health issue, particu-
larly in Mediterranean countries such as Greece. Although parental influences on children’s
weight have been extensively studied, fewer studies have jointly examined parental mental
health, feeding practices, sociodemographic factors, and biological stress markers. This
study aimed to investigate associations between psychological status, educational level,
feeding behaviors, and children’s Body Mass Index (BMI) in a Greek sample. A pilot assess-
ment of salivary cortisol was included in evaluating its feasibility as an objective biomarker
of parental stress. Subjects and Methods: A total of 103 parent–child dyads participated in
this cross-sectional study. Children’s BMI was classified using World Health Organization
(WHO) growth standards. Parental stress, anxiety, and depressive symptoms were assessed
using the Perceived Stress Scale-14 (PSS-14) and the Depression Anxiety Stress Scale-21
(DASS-21) questionnaires. Feeding practices were evaluated with the Comprehensive
Feeding Practices Questionnaire (CFPQ). Statistical analyses included Pearson correlations,
independent samples t-tests, one-way ANOVA, Mann–Whitney U, and Kruskal–Wallis
tests. A subsample provided saliva samples for cortisol analysis to assess feasibility and
explore the potential associations with parental stress indicators. Results: Parental BMI
showed a strong positive association with child BMI (p= 0.002). Higher parental anxiety
(p= 0.002) and depression (p= 0.009) were also associated with increased child BMI. Re-
strictive (p< 0.001) and emotion-driven (p< 0.001) feeding practices were associated with
higher child BMI, whereas monitoring (p= 0.013) and health-promoting feeding practices
(p= 0.001) appeared protective. Lower parental education was related to a higher BMI in
both parents (p= 0.001) and children (p= 0.002) and to more frequent use of restrictive
feeding strategies (p= 0.001). WHO charts identified a greater proportion of children
as overweight or obese compared with the Centers for Disease Control and Prevention
(CDC) criteria. The analysis showed statistically significant differences between the two
classification systems (
χ2
(4) = 159.704, p< 0.001), indicating that BMI categorization varies
considerably depending on the reference system used. No significant associations were
observed with residential environment or salivary cortisol, likely due to the limited size
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Nutrients 2026,18, 364 2 of 18
of the pilot biomarker subsample. Conclusions: The findings highlight the combined
effect of parental mental health status, educational level, and feeding practices on child
BMI within the Greek context. The preliminary inclusion of a biological stress marker
provides added value to the existing research in this area. These results underscore the
importance of prevention strategies that promote parental psychological wellbeing and
responsive feeding practices while addressing socioeconomic disparities to reduce the
childhood obesity risk.
Keywords: childhood obesity; parental stress; feeding practices; body mass index (BMI);
psychosocial determinants; salivary cortisol; World Health Organization (WHO); Greece
1. Introduction
Childhood obesity has emerged as one of the most urgent global public health chal-
lenges, with long-term consequences for children’s physical, psychological, and metabolic
health [
1
]. According to the World Health Organization (WHO), the worldwide prevalence
of overweight and obesity among children and adolescents has dramatically risen in recent
years, affecting millions of youths across low-, middle-, and high-income countries [
2
4
].
Europe has experienced similar upward trajectories, with Southern European countries
reporting the highest burden [
5
]. Greece consistently ranks among the countries with the
highest prevalence of childhood overweight and obesity, with national surveillance systems
indicating persistently elevated rates that render effective, targeted, evidence-based public
health approaches of utmost importance.
Children’s eating behaviors and weight evolution are shaped largely within the family
environment, where parental roles, household dynamics, and dietary routines exert profound
influence [
6
]. Parental feeding practices—defined as the strategies parents use to manage,
guide, or regulate their child’s eating—represent a key component of this environment [
7
].
These practices are not simply behaviorally driven; they are strongly shaped by parental
psychology, stress levels, cultural expectations, and socioeconomic pressures [
8
10
]. Accumu-
lating evidence suggests that parental stress plays a key role in modulating feeding behaviors,
often in ways that may compromise children’s ability to self-regulate intake and maintain a
healthy weight [
11
13
]. Stress can arise from multiple adversities, including financial strain,
professional demands, caregiving burdens, family conflict, and limited social support, all of
which may modify the consistency, responsiveness, and emotional status of parents’ feeding
interactions with their children [14].
Research has shown that higher levels of parental stress are associated with less struc-
tured and less responsive feeding practices, such as increased pressure to eat, inconsistent
limit setting, emotional feeding, greater use of food as reward or comfort, and reduced
monitoring of unhealthy foods [
15
18
]. These non-responsive practices have been linked
to disinhibited eating, emotional overeating, and diminished satiety responsiveness in
children, thereby contributing to excess weight gain over time [
19
21
]. Conversely, re-
sponsive feeding—characterized by supportive guidance, modeling healthy choices, and
providing autonomy—has been consistently associated with healthier dietary habits and
more optimal weight trajectories during childhood [2224].
Stress is a complex biopsychosocial process involving neuroendocrine, cognitive,
emotional, and behavioral pathways [
25
]. Central to the biological stress response is the
activation of the hypothalamic–pituitary–adrenal (HPA) axis, which leads to the release
of glucocorticoids, primarily cortisol [
26
]. Chronic or dysregulated cortisol secretion
has been associated with altered appetite control, preferences for energy-dense foods,
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Nutrients 2026,18, 364 3 of 18
increased reward-driven eating, abdominal adiposity, reduced physical activity, sleep
disturbances, and disruptions in metabolic homeostasis—pathways all implicated in obesity
development [
27
30
]. Stress-related physiological changes can also affect gastrointestinal
function, systemic inflammation, and energy balance, further supporting the link between
chronic stress exposure and weight gain in both adults and children [3133].
Within the family context, parental stress has both direct and indirect implications for
children’s health. Increased parental stress can amplify children’s own physiological and
emotional reactivity, diminish parental emotional availability, reduce positive communica-
tion, increase irritability or inconsistency in parenting, and contribute to more conflictual
or chaotic home environments [
34
37
]. These conditions may limit opportunities for struc-
tured family meals, reduce shared physical activity, increase children’s sedentary screen
time, and weaken routines that support healthy behaviors [
38
40
]. Parental stress may
further aggravate parental mental health, including symptoms of anxiety and depression,
which may provoke difficulties in maintaining supportive feeding practices or establish-
ing predictable household routines [
38
,
41
,
42
]. Co-parenting quality further moderates
these processes, with supportive and cooperative parenting acting as a buffer against
stress-related disruptions in family functioning [43].
Given these complex interactions, parental feeding practices represent a critical mech-
anism linking stress to children’s dietary behaviors and weight outcomes. The Comprehen-
sive Feeding Practices Questionnaire (CFPQ) and similar validated tools have been widely
used to assess the multidimensional aspects of feeding, including positive practices (e.g.,
monitoring, modeling, healthy-eating guidance) and maladaptive strategies (e.g., pressure
to eat, restriction, emotional feeding, food reward) [
44
46
]. Evidence consistently indicates
that responsive and autonomy-supportive feeding promotes healthier eating behaviors and
sustainable self-regulation of energy intake among children [
47
49
]. In contrast, coercive or
emotionally driven practices may undermine children’s satiety cues, promote overeating,
and contribute to unhealthy weight gain [5052].
In parallel with these family-level dynamics, epidemiological evidence underscores
the growing global and national burden of childhood obesity. Worldwide, the prevalence
of overweight and obesity among school-aged children has increased more than eight-fold
from 1975 to 2016, with substantial rises across regions regardless of economic develop-
ment [
53
55
]. Socioeconomic inequalities further exacerbate these trends, as children from
disadvantaged households face disproportionate exposure to obesogenic environments,
reduced access to healthy foods, and greater psychosocial stress [
56
58
]. In Greece, national
surveys such as the World Health Organization (WHO) European Childhood Obesity
Surveillance Initiative (COSI) and the Greek Examination of Cohorts (GRECO) Study have
consistently reported overweight and obesity rates exceeding 35–40% among primary
school children, among the highest in Europe [
7
,
59
,
60
]. These alarming trends reflect the
interplay between biological predispositions, obesogenic environments, family behaviors,
sociocultural norms, and psychosocial determinants including parental stress and mental
health [6163].
In the Greek context, childhood obesity is closely linked to family-centered lifestyles,
strong parental involvement in child feeding, and sociocultural norms that emphasize
shared meals and caregiving roles. At the same time, prolonged economic strain and
social stressors in recent decades may further influence psychological wellbeing and feed-
ing behaviors, highlighting the importance of examining these associations within a na-
tional framework.
Despite extensive research linking stress, mental health, and obesity, fewer studies
have thoroughly examined how parental psychological functioning interacts with feed-
ing practices, sociodemographic characteristics, and children’s anthropometric outcomes
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Nutrients 2026,18, 364 4 of 18
within the same analytical framework [
64
66
]. Even fewer studies have explored these
relationships in Mediterranean populations, where traditional dietary habits coexist with
rapid lifestyle changes, high obesity prevalence, and unique familial caregiving dynam-
ics [
53
,
67
,
68
]. The integration of biological stress markers, such as salivary cortisol, into
family-based pediatric obesity research remains limited, although such biomarkers pro-
vide valuable objective insight into the physiological stress pathways that may influence
parenting behavior and child health [69,70].
Although childhood obesity has been extensively studied, existing research has pre-
dominantly examined parental psychological factors, feeding practices, and sociodemo-
graphic characteristics as separate determinants of child weight outcomes. Fewer studies
have approached these domains in an integrated manner, particularly within Mediterranean
populations, where cultural, familial, and social contexts may shape parental behaviors
differently. Moreover, evidence remains limited regarding the concurrent consideration of
behavioral and biological indicators of stress in relation to childhood obesity.
The present study addresses these gaps by investigating the associations among parental
stress, parental mental health, feeding practices, and children’s weight status in a Greek sample
of parent–child dyads. By incorporating both psychosocial assessments and a preliminary
biological measure of parental stress (salivary cortisol), this research offers an integrated
examination of the behavioral, psychological, and physiological pathways that may contribute
to childhood obesity within contemporary Mediterranean contexts [
71
,
72
]. Understanding
these interconnected determinants is essential for informing family-centered interventions
and public health strategies aimed at mitigating the childhood obesity epidemic.
Based on the existing evidence, it was hypothesized that higher levels of parental
stress and poorer parental mental health would be associated with less responsive feeding
practices and higher child Body Mass Index. In addition, maladaptive feeding practices
were expected to be positively associated with children’s weight status. These hypotheses
were examined within the context of relevant sociodemographic characteristics.
2. Subjects and Methods
2.1. Study Design and Ethical Approval
This cross-sectional study was conducted between 2024 and 2025 in community set-
tings in the regions of Attica and Corinthia, as well as at the Pediatric Obesity Clinic of the
First Department of Pediatrics, Medical School of the National and Kapodistrian Univer-
sity of Athens (NKUA), at “Aghia Sofia” Children’s Hospital, Athens, Greece. The study
adhered to the ethical standards described in the Declaration of Helsinki.
The study protocol was approved by the Scientific Committee of the “Aghia Sofia”
Children’s Hospital (initial approval: protocol no. 19998/05.08.2024, approved on
11 October 2024; protocol amendment: protocol no. 13710/05.06.2025, approved on 17
June 2025). Written informed consent was obtained from all participating parents or legal
guardians prior to data collection.
2.2. Procedure
Parents were invited to participate either in community settings or during visits to the
Pediatric Obesity Clinic. After receiving standardized information about the study aims and
procedures, written informed consent was obtained from all participating parents or legal
guardians. Parents completed paper-based questionnaires, including a sociodemographic
form and validated instruments assessing perceived stress, mental health, and feeding
practices (approximately 20 min). Children’s anthropometric data were recorded as part
of the study assessment. In a pilot subsample, salivary cortisol samples were collected
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following standardized instructions and analyzed in the affiliated laboratory. All data were
coded and handled confidentially in accordance with ethical and data protection principles.
All psychometric instruments used in this study (PSS-14, DASS-21, and CFPQ) were
administered in their officially translated and validated Greek versions. Prior to data col-
lection, written permission was obtained from the respective developers and/or copyright
holders of each instrument.
2.3. Participants
The study included 103 parent–child dyads, consisting of 23 father–child dyads and
80 mother–child dyads, with children aged 2–12 years. Participants were recruited either
during clinic visits or through community outreach.
Inclusion Criteria
Parents of children aged 2–12 years.
Group A: Children with normal weight (5th–85th percentile) or underweight
(<5th percentile).
Group B: Children with overweight (>85th percentile), obesity (>95th percentile), or
severe obesity based on age- and sex-specific BMI percentiles.
Exclusion Criteria
Secondary causes of obesity (e.g., endocrine disorders such as Cushing syndrome or
growth hormone deficiency).
Genetic syndromes associated with abnormal weight (e.g., Down syndrome, Prader
Willi syndrome, etc.).
Chronic diseases or severe emotional/behavioral disorders.
Participant Flow
A total of 130 parents were approached; after applying inclusion and exclusion criteria,
103 dyads were included in the final analysis.
Anthropometric Measurements
Child height and weight were obtained either by trained clinical staff at the Pediatric
Obesity Clinic or parent-reported for community participants following standardized
written instructions.
BMI was calculated as weight (kg)/height (m2).
For ages 2–5 years, BMI-for-age z-scores were computed using the WHO child growth
standards (2006) [73] and WHO Anthro software (version 3.2.2) [74].
For ages 5–12 years, BMI-for-age z-scores were computed using the WHO growth
reference (2007) [75] and WHO AnthroPlus software (version 1.0.4) [76].
For cross-method comparability, BMI categories were additionally verified using the
CDC Child and Teen BMI Calculator [77].
2.4. Measures
Sociodemographic Questionnaire
A structured questionnaire was used to collect information on parental sex, age,
education, occupation, BMI, residence, and child characteristics (age, sex, height, weight,
health history).
Perceived Stress Scale (PSS-14)
Parental perceived stress was assessed using the Greek-validated version of the Per-
ceived Stress Scale-14 (PSS-14) [
78
], originally developed by Cohen et al. [
79
], a 14-item
measure evaluating the frequency of stress-related thoughts and feelings on a 0–4 Likert
scale, yielding total scores from 0 to 56. Higher total scores indicate higher perceived stress.
Depression Anxiety Stress Scale (DASS-21)
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The Greek-validated Depression Anxiety Stress Scale-21 (DASS-21) [
80
], originally
developed by Lovibond et al. [
81
], was used to assess symptoms of depression, anxiety,
and stress. The scale includes 21 items rated on a 0–3 Likert scale and produces three 7-item
subscale scores. Separate scores were calculated for each subscale (Depression, Anxiety,
and Stress), with higher scores indicating greater symptom severity.
Comprehensive Feeding Practices Questionnaire (CFPQ)
Parent feeding practices were measured using the Greek-validated Comprehensive
Feeding Practices Questionnaire (CFPQ) [
82
], originally developed by Musher-Eizenman
et al. [17], consisting of 42 items across six factors:
1. Healthy Eating Guidance (14 items);
2. Emotion Regulation/Food as Reward (6 items);
3. Monitoring (4 items);
4. Child Control (5 items);
5. Pressure (3 items);
6. Restriction (10 items).
Subscale scores were calculated by summing item responses within each factor, with
higher scores reflecting greater use of the corresponding feeding practice.
Pilot Salivary Cortisol Assessment
A subsample of 13 parents provided four salivary cortisol samples (awakening,
+30–45 min, evening, midnight) collected using Salivette
®
(Sarstedt, Nümbrecht, Ger-
many) devices. Samples were centrifuged, stored at
20
C, and analyzed in batch
using a validated immunoassay at the Clinical and Translational Endocrinology Labo-
ratory, “Aghia Sofia” Children’s Hospital. This assessment served as a feasibility pilot
to explore the biological markers of parental stress.
2.5. Statistical Analysis
Quantitative variables are presented as means
±
standard deviation or medians
(interquartile range), depending on distribution. Categorical variables are presented as
frequencies (%). Normality was assessed using the Kolmogorov–Smirnov test. Group
comparisons were conducted using independent sample t-tests and one-way ANOVA
for normally distributed variables, and Mann–Whitney U and Kruskal–Wallis tests for
non-normally distributed variables.
Associations between continuous variables were examined using Pearson correlation
coefficients. Chi-square tests were used to compare BMI classification between WHO and
CDC criteria. The significance level was set at p< 0.05. Analyses were performed using
IBM SPSS Statistics, version 29.
3. Results
3.1. Participants’ Characteristics
Table 1presents the demographic and psychological characteristics of the 103 par-
ticipating parent–child dyads. Most parents were women (77.7%), married (88.3%), and
residents of urban areas (67.0%). Regarding parental weight status, 5.8% of parents were
underweight, 40.8% had normal weight, while 35.9% were overweight and 17.5% were
classified as obese. The educational level was relatively high, with more than 70% of
parents having completed higher or postgraduate education.
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Table 1. Demographic characteristics and psychological status of parents (n= 103).
Variable Category n(%)
Parental sex Male 23 (22.3)
Female 80 (77.7)
Parental BMI category Underweight (<18.5) 6 (5.8)
Normal weight (18.5–24.9) 42 (40.8)
Overweight (25.0–29.9) 37 (35.9)
Obese (30.0) 18 (17.5)
Residence Urban area (Athens) 69 (67.0)
Non-urban area (Corinthia) 34 (33.0)
Marital status Single/divorced/widowed 12 (11.7)
Married 91 (88.3)
Educational level High school or lower 13 (12.6)
Vocational/technical training 16 (15.5)
University degree 34 (33.0)
Master’s degree 30 (29.1)
Doctoral degree 10 (9.7)
Occupation Other occupation 17 (16.5)
Public sector employee 26 (25.2)
Private sector employee 41 (39.8)
Self-employed 19 (18.4)
Child sex Boy 46 (44.7)
Girl 57 (55.3)
Child BMI (WHO) Underweight
(3SD Z < 2SD) 5 (4.9)
Normal weight
(1SD Z+1SD) 50 (48.5)
Overweight (+1SD < Z < +2SD) 18 (17.5)
Obese (+2SD Z+3SD) 11 (10.7)
Severely obese (Z > +3SD) 19 (18.4)
Child BMI (CDC) Underweight 9 (8.7)
Normal weight 49 (47.6)
Overweight 16 (15.5)
Obese 14 (13.6)
Severely obese 15 (14.6)
Parental stress (DASS-21
Stress subscale) Normal (0–14) 71 (68.9)
Mild (15–18) 12 (11.7)
Moderate (19–25) 12 (11.7)
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Table 1. Cont.
Variable Category n(%)
Severe (26–33) 6 (5.8)
Extreme (34+) 2 (1.9)
Parental anxiety
(DASS-21 Anxiety
subscale)
Normal (0–7) 75 (72.8)
Mild (8–9) 6 (5.8)
Moderate (10–14) 8 (7.8)
Severe (15–19) 6 (5.8)
Extreme (20+) 8 (7.8)
Parental depression
(DASS-21 Depression
subscale)
Normal (0–9) 78 (75.7)
Mild (10–13) 10 (9.7)
Moderate (14–20) 10 (9.7)
Severe (21–27) 3 (2.9)
Extreme (28+) 2 (1.9)
Note: BMI = Body Mass Index; WHO = World Health Organization; CDC = Centers for Disease Control and
Prevention; DASS-21 = Depression Anxiety Stress Scale-21.
Among the children, 55.3% were girls and 44.7% boys. According to the World Health
Organization (WHO) growth standards [
73
], 4.9% of children were classified as under-
weight, 48.5% had normal weight, 17.5% were overweight, 10.7% were obese, and 18.4%
were classified as severely obese. Similar distributions were observed using the Centers for
Disease Control and Prevention (CDC) criteria. Regarding parental psychological status
assessed with the Depression Anxiety Stress Scale-21 (DASS-21), the majority of parents
reported scores within the normal range for stress (68.9%), anxiety (72.8%), and depression
(75.7%). The prevalence of severe or very severe symptomatology was low across all three
subscales, remaining below 8% for each domain.
3.2. Correlations Between Parental Factors and Child BMI
Table 2summarizes the correlations between parental characteristics and children’s
BMI (WHO classification). Parental BMI showed a significant positive association with
child BMI (r = 0.304, p= 0.002), indicating that higher parental weight status was correlated
with higher child BMI values.
Child BMI was also positively associated with parental anxiety (r = 0.297, p= 0.002)
and depression (r = 0.255, p= 0.009), suggesting that parental psychological distress may
play a role in children’s weight trajectory.
Regarding feeding practices (CFPQ; Comprehensive Feeding Practices Questionnaire),
restrictive strategies (r = 0.558, p< 0.001), emotional feeding/food as reward (r = 0.466,
p< 0.001), and child control (r = 0.278, p= 0.004) were positively associated with higher
child BMI. Conversely, monitoring (r =
0.244, p= 0.013), pressure to eat (r =
0.204,
p= 0.039), and healthy eating guidance (r =
0.318, p= 0.001) were negatively associated
with child BMI.
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Table 2. Correlations between parental variables and child Body Mass Index (BMI) based on World
Health Organization (WHO) standards (n= 103).
Variable r p
Parental BMI 0.304 0.002 **
Perceived parental stress (PSS-14) 0.076 0.444
Parental stress (DASS-21 Stress subscale) 0.046 0.646
Parental anxiety (DASS-21 Anxiety subscale) 0.297 0.002 **
Parental depression (DASS-21 Depression subscale) 0.255 0.009 **
Monitoring (CFPQ subscale) 0.244 0.013 *
Child control (CFPQ subscale) 0.278 0.004 **
Emotional feeding/food as reward (CFPQ subscale) 0.466 0.000 **
Pressure to eat (CFPQ subscale) 0.204 0.039 *
Restriction (CFPQ subscale) 0.558 0.000 **
Healthy eating guidance (CFPQ subscale) 0.318 0.001 **
Cortisol AUCg (salivary) 0.168 0.584
Cortisol AUCi (salivary) 0.163 0.594
Note: * p< 0.05; ** p< 0.01. Pearson correlation coefficients were used. CFPQ = Comprehensive Feeding
Practices Questionnaire.
No significant correlations were found between child BMI and salivary cortisol indices
(AUCg, AUCi).
3.3. Associations Between Parental BMI, Psychological Factors, and Feeding Practices
Table 3displays the relationship between parental BMI, psychological status, and
feeding practices. Higher parental BMI was associated with greater perceived stress
(r = 0.196, p= 0.048), anxiety (r = 0.352, p< 0.001), depression (r = 0.262, p= 0.008), and
more restrictive feeding (r = 0.310, p= 0.001).
Table 3. Correlations between parental psychological factors and child feeding practices (n= 103).
Variable 1 Variable 2 r p
Parental BMI Perceived parental
stress (PSS-14) 0.196 0.048 *
Parental BMI
Parental anxiety
(DASS-21 Anxiety
subscale)
0.352 0.000 **
Parental BMI
Parental depression
(DASS-21 Depression
subscale)
0.262 0.008 **
Parental BMI Restriction (CFPQ) 0.310 0.001 **
Parental stress (DASS-21
Stress subscale) Child control (CFPQ) 0.218 0.027 *
Parental anxiety (DASS-21
Anxiety subscale)
Emotional
feeding/food as
reward (CFPQ)
0.307 0.002 **
Parental anxiety (DASS-21
Anxiety subscale) Restriction (CFPQ) 0.218 0.027 *
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Table 3. Cont.
Variable 1 Variable 2 r p
Parental depression (DASS-21
Depression subscale) Monitoring (CFPQ) 0.215 0.029 *
Parental depression (DASS-21
Depression subscale)
Emotional
feeding/Food as
reward (CFPQ)
0.199 0.044 *
Parental depression (DASS-21
Depression subscale)
Healthy eating
guidance (CFPQ) 0.331 0.001 **
Note: * p< 0.05; ** p< 0.01. Pearson correlation coefficients were used. CFPQ = Comprehensive Feeding
Practices Questionnaire.
Parental stress (DASS-21) was positively correlated with child control feeding prac-
tices (r = 0.218, p= 0.027). Parental anxiety was associated with both emotional feeding
(r = 0.307, p= 0.002) and restriction (r = 0.218, p= 0.027). Depression correlated negatively
with monitoring (r =
0.215, p= 0.029) and healthy eating guidance (r =
0.331, p= 0.001)
and positively with emotional feeding (r = 0.199, p= 0.044).
3.4. Differences by Parental Education Level
As shown in Table 4, parental education was significantly related to parental BMI
(p= 0.001), child BMI (p= 0.002), and restrictive feeding practices (p= 0.001). Parents with
lower educational attainment (high school graduates, graduates from technical schools) had
higher BMI (p= 0.001), children with higher BMI (p= 0.002), and used more restrictive feeding
practices (p= 0.001) compared with parents holding postgraduate or doctoral degrees.
Table 4. Differences in parental education level across parental and child outcomes (n= 103).
Education Level Parental BMI
(Mean ±SD)
Child BMI WHO
(Mean ±SD)
Food Restriction
(CFPQ) (Mean ±SD)
Perceived Parental
Stress (PSS-14)
(Mean ±SD)
High school or lower
(n= 13) 29.04 ±6.72 †,ˆ 19.19 ±4.41 †,ˆ 31.08 ±5.77 †,ˆ 25.62 ±5.68
Vocational/technical
training (n= 16) 28.94 ±6.77 #,< 22.83 ±6.30 #,< 34.94 ±8.58 #,< 28.75 ±8.61
University
degree—TEI/AEI
(n= 34)
25.53 ±4.80 20.20 ±5.52 28.53 ±9.76 22.65 ±8.47
Master’s degree
(n= 30) 24.52 ±5.10 ˆ,< 16.99 ±2.92 ˆ,< 25.13 ±7.58 ˆ,< 22.30 ±5.96
Doctoral degree
(n= 10) 21.91 ±4.25 ,# 16.42 ±4.14 †,# 20.30 ±7.15 †,# 22.20 ±8.55
p-value (Significance) 0.001 ** 0.002 ** 0.001 ** 0.05 *
Note: * p< 0.05; ** p< 0.01. One-way ANOVA and Kruskal–Wallis with post hoc pairwise comparisons were
conducted to assess differences across education groups. Symbols denote statistically significant pairwise
differences:
significantly different from High school or lower, # significantly different from IEK, ˆ significantly
different from University degree, < significantly different from Doctoral degree.
3.5. Differences by Parental Gender
Table 5presents comparisons between mothers and fathers. Mann–Whitney U tests
were used due to non-normal variable distributions. Fathers had significantly higher BMIs
than mothers (p= 0.006). Mothers reported significantly greater monitoring of their child’s
eating (p= 0.020). No significant gender differences were observed for stress, anxiety,
depression, or most feeding practice subscales.
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Table 5. Differences in parental variables according to parental sex (n= 103).
Variable Men
(Median, IQR)
Women
(Median, IQR)
p
(Two-Tailed)
Parental BMI 28 (4) 25 (4) 0.006 *
Child BMI (WHO) 49 (8) 53 (6) 0.587
Perceived parental stress (PSS-14) 24 (8) 24 (6) 0.651
Parental stress
(DASS-21 Stress subscale) 50 (8) 53 (7) 0.721
Parental anxiety
(DASS-21 Anxiety subscale) 48 (8) 53 (8) 0.438
Parental depression (DASS-21
Depression subscale) 48 (7) 53 (6) 0.511
Monitoring (CFPQ) 15 (3) 17 (4) 0.020 *
Child control (CFPQ) 49 (6) 53 (6) 0.535
Emotional feeding/food as reward
(CFPQ) 57 (7) 51 (7) 0.402
Pressure to eat (CFPQ) 62 (7) 49 (8) 0.059
Healthy eating guidance (CFPQ) 44 (8) 54 (8) 0.155
Note: * p< 0.05. Differences were assessed using the independent samples t-test and Mann–Whitney U test
(two-tailed). CFPQ = Comprehensive Feeding Practices Questionnaire; BMI = Body Mass Index.
3.6. Differences by Child Gender
According to Table 6, parents of boys had significantly higher BMIs than parents of
girls (p= 0.006). Parents of girls demonstrated higher monitoring of their children’s dietary
behaviors (p= 0.020). All other variables, including child BMI, parental stress, anxiety, and
depression, did not significantly differ between boys and girls.
Table 6. Comparison of boys and girls on key variables (n= 103).
Variable Boys
(Median, IQR)
Girls
(Median, IQR)
p
Child BMI (WHO) 16.2 (2.5) 15.7 (2.6) 0.152
Parental BMI 28 (4.7) 25 (6.7) 0.006 *
Perceived parental stress (PSS-14) 24 (12) 24 (12) 0.651
Parental stress
(DASS-21 Stress subscale) 50 (10) 53 (8) 0.721
Parental anxiety
(DASS-21 Anxiety subscale) 48 (11) 53 (9) 0.438
Parental depression (DASS-21
Depression subscale) 48 (9) 53 (8) 0.511
Monitoring (CFPQ) 15 (3) 17 (3) 0.020 *
Child control (CFPQ) 49 (7) 53 (6) 0.535
Emotional feeding/food as reward
(CFPQ) 57 (10) 51 (7) 0.402
Pressure to eat (CFPQ) 62 (7) 49 (7) 0.059
Healthy eating guidance (CFPQ) 44 (7) 54 (7) 0.155
Note: * p< 0.05. Comparisons were performed using the independent samples t-tests and Mann–Whitney U test
(two-tailed). CFPQ = Comprehensive Feeding Practices Questionnaire; BMI = Body Mass Index.
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Nutrients 2026,18, 364 12 of 18
3.7. Comparison Between WHO and CDC BMI Classification
Table 7compares BMI categorization based on WHO versus CDC criteria for
94 children. Of the initial 103 participants, 94 were included because the “underweight”
category contained a limited number of cases and was excluded from statistical analysis.
WHO identified a slightly higher proportion of overweight and obese children compared
with CDC charts. The analysis showed statistically significant differences between the two
classification systems (
χ2
(4) = 159.704, p< 0.001), indicating that BMI categorization varies
considerably depending on the reference system used.
Table 7. Comparison of child BMI classification based on WHO and CDC standards.
BMI Category WHO n(%) CDC n(%)
Normal weight 46 (48.9) 49 (52.1)
Overweight 18 (19.1) 16 (17.0)
Obese 30 (31.9) 29 (30.9)
Total 94 (100) 94 (100)
Note: Data are presented as absolute numbers (n) and percentages (%). BMI categories were defined using World
Health Organization (WHO) and Centers for Disease Control and Prevention (CDC) growth standards.
4. Discussion
This study examined the associations between parental psychological functioning,
feeding practices, sociodemographic characteristics, and children’s BMI in a Greek sample
of 103 parent–child pairs. Moreover, a pilot study that included 13 parents, investigating
the use of salivary cortisol as a biological stress marker, was also conducted in a small
number of participants. Overall, the findings support the multifactorial nature of childhood
obesity and highlight the central role of the family environment, particularly parental
mental health and feeding practices, in shaping children’s weight outcomes [6163].
Consistent with the extensive international evidence, parental BMI was strongly as-
sociated with child BMI, reinforcing the well-established intergenerational transmission
of obesity risk. This link reflects both shared genetic predispositions and shared behav-
ioral and environmental factors within families [
29
,
53
,
54
]. Notably, parental anxiety and
depression were also positively related to child BMI. These results suggest that beyond
behavioral modeling, the emotional milieu of the household may influence children’s eating
patterns and weight status. Previous studies have reported similar associations, indicating
that psychological distress can disrupt the parental capacity for consistent and responsive
feeding practices and may contribute to unstructured food environments [1,11,12].
Feeding practices demonstrated clear and meaningful associations with children’s
BMI. Restrictive feeding, emotional feeding, and greater child control were positively
associated with higher child BMI, whereas monitoring and healthy eating guidance were
inversely associated. These findings align with previous research showing that restrictive
and emotionally driven strategies may undermine children’s ability to self-regulate food
intake, increase the desirability of high-fat, high-calorie foods, or encourage maladaptive
emotional eating behaviors [
18
,
19
,
52
]. In contrast, responsive practices—such as encourag-
ing healthy eating and monitoring unhealthy food intake—have been linked to healthier
dietary patterns and lower obesity risk [
15
,
22
,
47
]. Notably, the observed patterns also
mirror cultural dynamics in Mediterranean settings, where food is often intertwined with
emotional expression, reward, and parental care [53,67,68].
Educational level emerged as a strong determinant of parental and child weight status
as well as feeding practices. Parents with lower educational attainment exhibited higher
BMIs, had children with higher BMIs, and used more restrictive feeding practices. These
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Nutrients 2026,18, 364 13 of 18
gradients have already been documented in numerous European cohorts and reflect broader
socioeconomic inequalities regarding access to health information, nutrition literacy, and
lifestyle opportunities [
56
58
]. In Greece, where disparities in health literacy remain
prominent, these results underscore the relevance of social determinants in shaping obesity
risk [7,59,60].
The comparison between the WHO and CDC classification systems revealed that
WHO charts identified a greater proportion of children as overweight or obese. This is
consistent with the existing literature reporting the higher sensitivity of WHO references,
which are based on optimal growth standards rather than population-based norms [
53
,
54
].
For clinical and public health practices, this finding highlights the importance of selecting a
classification system aligned with the aims of screening and early identification of at-risk
pediatric populations.
No significant associations were detected between salivary cortisol indices and psy-
chological or behavioral variables. This is likely attributable to the limited number of
samples obtained during the pilot phase, which severely constrained the statistical power.
However, the successful implementation of the saliva collection protocol supports the
feasibility of this research approach in future obesity studies. Incorporating biological
stress markers may be particularly valuable in stress-related obesity research, providing
a measurable biological marker to assess the impact of stress on human behavior and its
consequences [69,70].
Overall, the findings point to the importance of addressing parental mental health in
the context of childhood obesity prevention frameworks. Interventions that support parents
in managing their own stress, enhancing emotional wellbeing, and adopting responsive
feeding practices may yield substantial benefits for children’s weight trajectories [
11
,
15
,
47
].
Health promotion programs in Greece should consider integrating parental psychosocial
support with traditional nutritional guidance, particularly among families with lower
educational attainment [
56
58
]. Future research should expand biomarker assessment,
incorporate qualitative approaches to capture cultural nuances in feeding practices, and
adopt longitudinal designs to disentangle causal mechanisms. By capturing the interplay
between psychological, behavioral, and social determinants, this study contributes to a
more comprehensive understanding of childhood obesity within the Greek context.
5. Strengths and Limitations
5.1. Strengths
This study simultaneously examined parental mental health, feeding practices, and
sociodemographic factors in relation to childhood BMI, offering a comprehensive biopsy-
chosocial perspective rarely addressed in Greek populations. The use of validated Greek
versions of all psychometric tools (PSS-14, DASS-21, CFPQ), standardized anthropometric
methods, and WHO/CDC growth references strengthens the robustness of the findings. An
additional strength is the pilot inclusion of salivary cortisol, which, although exploratory,
demonstrates the feasibility of integrating biological stress markers in family-based obe-
sity research.
5.2. Limitations
The cross-sectional design limits the assessment of causal inferences and the
sample—although diverse—was not nationally representative. Self-reported psychologi-
cal and behavioral measures may be influenced by reporting bias. The relatively small
biomarker subsample in the pilot study assessing salivary cortisol as a stress biomarker
limited the extraction of meaningful results. Finally, parental measurements collected at
home for part of the sample may have introduced measurement variability.
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Nutrients 2026,18, 364 14 of 18
In addition, the analyses were restricted to bivariate associations. Although multi-
variable models could further clarify independent predictors of child BMI, the present
study was designed as an exploratory investigation focusing on the initial relationships
between parental psychological factors, feeding practices, and child weight status. Future
studies with larger samples and longitudinal designs should incorporate multivariable
analytical approaches.
6. Conclusions
This study highlights the multifactorial nature of childhood obesity, demonstrating
that parental mental health, educational level, and feeding practices jointly influence
children’s BMI. Higher parental anxiety, depression, and restrictive or emotion-driven
feeding behaviors were associated with increased child BMI, whereas monitoring and
health-oriented guidance appeared protective. These findings underscore the need for
family-centered prevention strategies that support parental psychological wellbeing and
promote responsive, non-restrictive feeding approaches. Although the exploratory salivary
cortisol sub-study did not yield significant associations, its feasibility suggests potential
value for future biomarker-driven research. Further longitudinal and large-scale stud-
ies are required to clarify the causal pathways and inform more targeted public health
interventions in Greece and comparable settings.
Author Contributions: Conceptualization, V.S. and C.K.-G.; methodology, V.S., M.M., E.A. and X.T.;
formal analysis, V.S. and M.M.; investigation, V.S.; resources, C.K.-G.; data curation, V.S. and M.M.;
writing—original draft preparation, V.S.; writing—review and editing, Y.M., O.A., M.M., E.A., X.T.,
P.P., S.K. and C.K.-G.; visualization, V.S.; supervision, C.K.-G.; project administration, C.K.-G. All
authors have read and agreed to the published version of the manuscript.
Funding: This study received no external funding and was conducted as part of the requirements for
the MSc program “Science of Stress and Health Promotion” at the National and Kapodistrian University
of Athens.
Institutional Review Board Statement: The study was conducted in accordance with the Declaration
of Helsinki and has been approved by the Scientific Council and the Ethics and Deontology Committee
of “Aghia Sofia” Children’s Hospital, Athens, Greece. The initial protocol was approved on 11 October
2024 (approval number: 19998/05.08.2024) and subsequently amended and approved on 17 June
2025 (approval number: 13710/05.06.2025).
Informed Consent Statement: Written informed consent was obtained from all parents or legal
guardians involved in the study. All participants were fully informed about the study procedures,
objectives, confidentiality measures, and their right to withdraw at any time without consequences.
Data Availability Statement: The data supporting the findings of this study are not publicly available
due to ethical and privacy restrictions involving minors. De-identified datasets may be provided
by the corresponding author upon reasonable request and subject to approval by the institutional
ethics committee.
Acknowledgments: The first author, V. Stymfaliadi, would like to express her heartfelt gratitude to
her family for their continuous support during the preparation of this manuscript.
Conflicts of Interest: The authors declare no conflicts of interest.
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